Imagine a genetic mutation that causes troponin to bind to calcium much more strongly than usual,...

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Imagine a genetic mutation that causes troponin to bind to calciummuch more strongly than usual, i.e., once the troponin binds tocalcium, it is very slow to release the calcium again. What effectswould this have an effect on muscle contraction? (That is, wouldcontractions be stronger/weaker than normal? Would they last longerthan normal, or not as long?) Explain your reasoning.
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Troponin Tn is the sarcomeric Ca2 regulator for striated skeletal and cardiac muscle contraction On binding Ca2 Tn transmits information via structural changes throughout the actintropomyosin    See Answer
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